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- Thread starter BigWoof31
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Taosman
Your Cousin In New Mexxico
A "Rocky Ride"
"The world will be fighting coronavirus for the next three to four years as virus hot spots skip from nation to nation, and the pandemic's toll will linger for decades, said Dr. Larry Brilliant, a California epidemiologist who was part of a World Health Organization team in the 1970s that helped eradicate smallpox."
"But it's "not all doom and gloom," with effective vaccines likely to emerge from dozens of candidates worldwide and effective treatments, including convalescent plasma and monoclonal antibodies, to help people recover more quickly, said Brilliant, who chairs Ending Pandemics advisory board."
https://www.msn.com/en-us/news/us/w...d-19-turmoil-for-years/ar-BB17wGHU?li=BBnb7Kz
"The world will be fighting coronavirus for the next three to four years as virus hot spots skip from nation to nation, and the pandemic's toll will linger for decades, said Dr. Larry Brilliant, a California epidemiologist who was part of a World Health Organization team in the 1970s that helped eradicate smallpox."
"But it's "not all doom and gloom," with effective vaccines likely to emerge from dozens of candidates worldwide and effective treatments, including convalescent plasma and monoclonal antibodies, to help people recover more quickly, said Brilliant, who chairs Ending Pandemics advisory board."
https://www.msn.com/en-us/news/us/w...d-19-turmoil-for-years/ar-BB17wGHU?li=BBnb7Kz
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Not sure about the TP situation there...last I heard was that TP was available but stores were limiting purchases.
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This is a good sign, but it’s one step in a complicated immune response. It’s not certain that “they” even CAN make something of it, and it seems likely that several other avenues of research will make some difference before this turns into anything. It might eventually be a game changer, it might not, it’s not likely to be soon either way.
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Taosman
Your Cousin In New Mexxico
"How is it that six months into a respiratory pandemic, we still have so little guidance about this all-important variable, the very air we breathe?"
The dominance of a ballistic-droplet mode of transmission in this pandemic would mean that we should focus mostly on staying out of droplets’ range, especially to prevent them from falling on our unprotected mouth, nose, and eyes—hence the social-distancing guidelines. It also would mean that keeping that distance would be enough to stay safe from an infected person, on the other side of a room for example. (Of course, our hands can still potentially pick them up from surfaces and bring them to our face, hence the importance of hand-washing.)
https://www.theatlantic.com/health/...king-more-about-airborne-transmission/614737/
The dominance of a ballistic-droplet mode of transmission in this pandemic would mean that we should focus mostly on staying out of droplets’ range, especially to prevent them from falling on our unprotected mouth, nose, and eyes—hence the social-distancing guidelines. It also would mean that keeping that distance would be enough to stay safe from an infected person, on the other side of a room for example. (Of course, our hands can still potentially pick them up from surfaces and bring them to our face, hence the importance of hand-washing.)
https://www.theatlantic.com/health/...king-more-about-airborne-transmission/614737/
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I was gonna say....English please.
Oh, yeah sorry.
Its good. What its saying is, and I posted something related to this the other day, and its that there is Tcell resistance in people that were not exposed to covid 19 have this CD4 Tcell response (so I think, and someone can correct me) these are helper TCells which aid in immune system response from having learned (i this case) orher coronavirus are bad (common cold) - so some people have some previous protection based on this.. in the abstract 20-50%. This is on the same line as a German study I previously mentioned which had it at 35% (I think)
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sparcboxbuck
What happened to my ¤cash?
This. It’s similar enough to things that the body has fought off in the past that it’s triggering the response or has created an elevated level of protection — which I’m not sure. I’m following this topic specific to how it may be used in vaccines.
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By itself, this portion of the immune response might help ameliorate other coronavirus infectious, but it’s not clear that this will do anything to prevent the cytokine storms that are the beginning of the end for SARS-CoV-2’s victims. Admittedly I did not read this particular study all the way to the end, I’d love to be wrong here.
All coronaviruses evoke a 2-part immune response, of which this is part 1. The reason more of us don’t have this level of immunity is because this kind of immunity fades much faster than others. But some of those who are developing vaccines are working on ways of piggy-backing on residual immunity to seasonal coronavirus. This is where there is promise here.
Apart from the clues this gives the researchers, the upshot is this: if you’ve had a cold recently, there’s a 20% chance that you might have reduced symptoms from COVID-19. There is little chance that it will affect you’re chances of dying, but that risk isn’t great for most of you anyway.
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The above is a massive over simplification. Especially the bit about the 2-part response. Hopefully it’s good enough to be getting on with
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You're not wrong from the perspective if they they to make vaccines out of other coronaviruses and it only elicits a TCell response and not. Bcell response, indeed you won't have a very good vaccine.
That misses a couple other implications here around clinical outcomes and herd immunity modeling. Themes IbI have posting about for a long time in this thread.
But I mean I can stop posting and you can see interpret everything for us.
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